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Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs .

Journal of cardiovascular development and disease

Viswanathan MC, Blice-Baum AC, Sang TK, Cammarato A.
PMID: 27500162
J Cardiovasc Dev Dis. 2016 Jun;3(2). doi: 10.3390/jcdd3020019. Epub 2016 May 24.

Valosin-containing protein (VCP) is a highly conserved mechanoenzyme that helps maintain protein homeostasis in all cells and serves specialized functions in distinct cell types. In skeletal muscle, it is critical for myofibrillogenesis and atrophy. However, little is known about...

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Viswanathan MC, Blice-Baum AC, Sang TK, et al. Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs . J Cardiovasc Dev Dis. 2016;3(2)doi: 10.3390/jcdd3020019.
Viswanathan, M. C., Blice-Baum, A. C., Sang, T. K., & Cammarato, A. (2016). Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs . Journal of cardiovascular development and disease, 3(2), . https://doi.org/10.3390/jcdd3020019
Viswanathan, Meera C, et al. "Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs ." Journal of cardiovascular development and disease vol. 3,2 (2016). doi: https://doi.org/10.3390/jcdd3020019
Viswanathan MC, Blice-Baum AC, Sang TK, Cammarato A. Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs . J Cardiovasc Dev Dis. 2016 Jun;3(2). doi: 10.3390/jcdd3020019. Epub 2016 May 24. PMID: 27500162; PMCID: PMC4973812.
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Transcriptional Regulation of Heart Development in Zebrafish.

Journal of cardiovascular development and disease

Lu F, Langenbacher AD, Chen JN.
PMID: 27148546
J Cardiovasc Dev Dis. 2016 Apr 09;3(2). doi: 10.3390/jcdd3020014. Epub 2016 Apr 07.

Cardiac transcription factors orchestrate the complex cellular and molecular events required to produce a functioning heart. Misregulation of the cardiac transcription program leads to embryonic developmental defects and is associated with human congenital heart diseases. Recent studies have expanded...

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Lu F, Langenbacher AD, Chen JN. Transcriptional Regulation of Heart Development in Zebrafish. J Cardiovasc Dev Dis. 2016;3(2)doi: 10.3390/jcdd3020014.
Lu, F., Langenbacher, A. D., & Chen, J. N. (2016). Transcriptional Regulation of Heart Development in Zebrafish. Journal of cardiovascular development and disease, 3(2), . https://doi.org/10.3390/jcdd3020014
Lu, Fei, et al. "Transcriptional Regulation of Heart Development in Zebrafish." Journal of cardiovascular development and disease vol. 3,2 (2016). doi: https://doi.org/10.3390/jcdd3020014
Lu F, Langenbacher AD, Chen JN. Transcriptional Regulation of Heart Development in Zebrafish. J Cardiovasc Dev Dis. 2016 Apr 09;3(2). doi: 10.3390/jcdd3020014. Epub 2016 Apr 07. PMID: 27148546; PMCID: PMC4852312.
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Evidence of Aortopathy in Mice with Haploinsufficiency of .

Journal of cardiovascular development and disease

Koenig SN, Bosse KM, Nadorlik HA, Lilly B, Garg V.
PMID: 25914885
J Cardiovasc Dev Dis. 2015;2(1):17-30. doi: 10.3390/jcdd2010017.

Thoracic aortic aneurysms (TAA) are a significant cause of morbidity and mortality in humans. While the exact etiology is unknown, genetic factors play an important role. Mutations in

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Koenig SN, Bosse KM, Nadorlik HA, et al. Evidence of Aortopathy in Mice with Haploinsufficiency of . J Cardiovasc Dev Dis. 2015;2(1):17-30doi: 10.3390/jcdd2010017.
Koenig, S. N., Bosse, K. M., Nadorlik, H. A., Lilly, B., & Garg, V. (2015). Evidence of Aortopathy in Mice with Haploinsufficiency of . Journal of cardiovascular development and disease, 2(1), 17-30. https://doi.org/10.3390/jcdd2010017
Koenig, Sara N, et al. "Evidence of Aortopathy in Mice with Haploinsufficiency of ." Journal of cardiovascular development and disease vol. 2,1 (2015): 17-30. doi: https://doi.org/10.3390/jcdd2010017
Koenig SN, Bosse KM, Nadorlik HA, Lilly B, Garg V. Evidence of Aortopathy in Mice with Haploinsufficiency of . J Cardiovasc Dev Dis. 2015;2(1):17-30. doi: 10.3390/jcdd2010017. PMID: 25914885; PMCID: PMC4407710.
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Targeted .

Journal of cardiovascular development and disease

Judge DP, Neamatalla H, Norris RA, Levine RA, Butcher JT, Vignier N, Kang KH, Nguyen Q, Bruneval P, Perier MC, Messas E, Jeunemaitre X, de Vlaming A, Markwald R, Carrier L, Hagège AA.
PMID: 26819945
J Cardiovasc Dev Dis. 2015;2(2):48-65. doi: 10.3390/jcdd2020048. Epub 2015 Apr 21.

No abstract available.

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Judge DP, Neamatalla H, Norris RA, et al. Targeted . J Cardiovasc Dev Dis. 2015;2(2):48-65doi: 10.3390/jcdd2020048.
Judge, D. P., Neamatalla, H., Norris, R. A., Levine, R. A., Butcher, J. T., Vignier, N., Kang, K. H., Nguyen, Q., Bruneval, P., Perier, M. C., Messas, E., Jeunemaitre, X., de Vlaming, A., Markwald, R., Carrier, L., & Hagège, A. A. (2015). Targeted . Journal of cardiovascular development and disease, 2(2), 48-65. https://doi.org/10.3390/jcdd2020048
Judge, Daniel P, et al. "Targeted ." Journal of cardiovascular development and disease vol. 2,2 (2015): 48-65. doi: https://doi.org/10.3390/jcdd2020048
Judge DP, Neamatalla H, Norris RA, Levine RA, Butcher JT, Vignier N, Kang KH, Nguyen Q, Bruneval P, Perier MC, Messas E, Jeunemaitre X, de Vlaming A, Markwald R, Carrier L, Hagège AA. Targeted . J Cardiovasc Dev Dis. 2015;2(2):48-65. doi: 10.3390/jcdd2020048. Epub 2015 Apr 21. PMID: 26819945; PMCID: PMC4725593.
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Gene Therapy Approaches to Biological Pacemakers.

Journal of cardiovascular development and disease

Farraha M, Kumar S, Chong J, Cho HC, Kizana E.
PMID: 30347716
J Cardiovasc Dev Dis. 2018 Oct 19;5(4). doi: 10.3390/jcdd5040050.

Bradycardia arising from pacemaker dysfunction can be debilitating and life threatening. Electronic pacemakers serve as effective treatment options for pacemaker dysfunction. They however present their own limitations and complications. This has motivated research into discovering more effective and innovative...

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Farraha M, Kumar S, Chong J, et al. Gene Therapy Approaches to Biological Pacemakers. J Cardiovasc Dev Dis. 2018;5(4)doi: 10.3390/jcdd5040050.
Farraha, M., Kumar, S., Chong, J., Cho, H. C., & Kizana, E. (2018). Gene Therapy Approaches to Biological Pacemakers. Journal of cardiovascular development and disease, 5(4), . https://doi.org/10.3390/jcdd5040050
Farraha, Melad, et al. "Gene Therapy Approaches to Biological Pacemakers." Journal of cardiovascular development and disease vol. 5,4 (2018). doi: https://doi.org/10.3390/jcdd5040050
Farraha M, Kumar S, Chong J, Cho HC, Kizana E. Gene Therapy Approaches to Biological Pacemakers. J Cardiovasc Dev Dis. 2018 Oct 19;5(4). doi: 10.3390/jcdd5040050. PMID: 30347716; PMCID: PMC6306875.
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A-Kinase Anchoring Protein-Lbc: A Molecular Scaffold Involved in Cardiac Protection.

Journal of cardiovascular development and disease

Diviani D, Osman H, Reggi E.
PMID: 29419761
J Cardiovasc Dev Dis. 2018 Feb 08;5(1). doi: 10.3390/jcdd5010012.

Heart failure is a lethal disease that can develop after myocardial infarction, hypertension, or anticancer therapy. In the damaged heart, loss of function is mainly due to cardiomyocyte death and associated cardiac remodeling and fibrosis. In this context, A-kinase...

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Diviani D, Osman H, Reggi E. A-Kinase Anchoring Protein-Lbc: A Molecular Scaffold Involved in Cardiac Protection. J Cardiovasc Dev Dis. 2018;5(1)doi: 10.3390/jcdd5010012.
Diviani, D., Osman, H., & Reggi, E. (2018). A-Kinase Anchoring Protein-Lbc: A Molecular Scaffold Involved in Cardiac Protection. Journal of cardiovascular development and disease, 5(1), . https://doi.org/10.3390/jcdd5010012
Diviani, Dario, et al. "A-Kinase Anchoring Protein-Lbc: A Molecular Scaffold Involved in Cardiac Protection." Journal of cardiovascular development and disease vol. 5,1 (2018). doi: https://doi.org/10.3390/jcdd5010012
Diviani D, Osman H, Reggi E. A-Kinase Anchoring Protein-Lbc: A Molecular Scaffold Involved in Cardiac Protection. J Cardiovasc Dev Dis. 2018 Feb 08;5(1). doi: 10.3390/jcdd5010012. PMID: 29419761; PMCID: PMC5872360.
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Cardiac Fibroblasts and the Extracellular Matrix in Regenerative and Nonregenerative Hearts.

Journal of cardiovascular development and disease

Hortells L, Johansen AKZ, Yutzey KE.
PMID: 31434209
J Cardiovasc Dev Dis. 2019 Aug 20;6(3). doi: 10.3390/jcdd6030029.

During the postnatal period in mammals, the heart undergoes significant remodeling and cardiac cells progressively lose their embryonic characteristics. At the same time, notable changes in the extracellular matrix (ECM) composition occur with a reduction in the components considered...

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Hortells L, Johansen AKZ, Yutzey KE. Cardiac Fibroblasts and the Extracellular Matrix in Regenerative and Nonregenerative Hearts. J Cardiovasc Dev Dis. 2019;6(3)doi: 10.3390/jcdd6030029.
Hortells, L., Johansen, A. K. Z., & Yutzey, K. E. (2019). Cardiac Fibroblasts and the Extracellular Matrix in Regenerative and Nonregenerative Hearts. Journal of cardiovascular development and disease, 6(3), . https://doi.org/10.3390/jcdd6030029
Hortells, Luis, et al. "Cardiac Fibroblasts and the Extracellular Matrix in Regenerative and Nonregenerative Hearts." Journal of cardiovascular development and disease vol. 6,3 (2019). doi: https://doi.org/10.3390/jcdd6030029
Hortells L, Johansen AKZ, Yutzey KE. Cardiac Fibroblasts and the Extracellular Matrix in Regenerative and Nonregenerative Hearts. J Cardiovasc Dev Dis. 2019 Aug 20;6(3). doi: 10.3390/jcdd6030029. PMID: 31434209; PMCID: PMC6787677.
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Renal Venous Pattern: A New Parameter for Predicting Prognosis in Heart Failure Outpatients.

Journal of cardiovascular development and disease

Puzzovivo A, Monitillo F, Guida P, Leone M, Rizzo C, Grande D, Ciccone MM, Iacoviello M.
PMID: 30400289
J Cardiovasc Dev Dis. 2018 Nov 03;5(4). doi: 10.3390/jcdd5040052.

AIM OF THE STUDY: In chronic heart failure (CHF) patients, renal congestion plays a key role in determining the progression of renal dysfunction and a worse prognosis. The aim of this study was to define the role of Doppler...

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Puzzovivo A, Monitillo F, Guida P, et al. Renal Venous Pattern: A New Parameter for Predicting Prognosis in Heart Failure Outpatients. J Cardiovasc Dev Dis. 2018;5(4)doi: 10.3390/jcdd5040052.
Puzzovivo, A., Monitillo, F., Guida, P., Leone, M., Rizzo, C., Grande, D., Ciccone, M. M., & Iacoviello, M. (2018). Renal Venous Pattern: A New Parameter for Predicting Prognosis in Heart Failure Outpatients. Journal of cardiovascular development and disease, 5(4), . https://doi.org/10.3390/jcdd5040052
Puzzovivo, Agata, et al. "Renal Venous Pattern: A New Parameter for Predicting Prognosis in Heart Failure Outpatients." Journal of cardiovascular development and disease vol. 5,4 (2018). doi: https://doi.org/10.3390/jcdd5040052
Puzzovivo A, Monitillo F, Guida P, Leone M, Rizzo C, Grande D, Ciccone MM, Iacoviello M. Renal Venous Pattern: A New Parameter for Predicting Prognosis in Heart Failure Outpatients. J Cardiovasc Dev Dis. 2018 Nov 03;5(4). doi: 10.3390/jcdd5040052. PMID: 30400289; PMCID: PMC6306853.
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Cardiac Fibroblast to Myofibroblast Phenotype Conversion-An Unexploited Therapeutic Target.

Journal of cardiovascular development and disease

Czubryt MP.
PMID: 31426390
J Cardiovasc Dev Dis. 2019 Aug 16;6(3). doi: 10.3390/jcdd6030028.

Fibrosis occurs when the synthesis of extracellular matrix outpaces its degradation, and over time can negatively impact tissue and organ function. In the case of cardiac fibrosis, contraction and relaxation of the heart can be impaired to the point...

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Czubryt MP. Cardiac Fibroblast to Myofibroblast Phenotype Conversion-An Unexploited Therapeutic Target. J Cardiovasc Dev Dis. 2019;6(3)doi: 10.3390/jcdd6030028.
Czubryt, M. P. (2019). Cardiac Fibroblast to Myofibroblast Phenotype Conversion-An Unexploited Therapeutic Target. Journal of cardiovascular development and disease, 6(3), . https://doi.org/10.3390/jcdd6030028
Czubryt, Michael P. "Cardiac Fibroblast to Myofibroblast Phenotype Conversion-An Unexploited Therapeutic Target." Journal of cardiovascular development and disease vol. 6,3 (2019). doi: https://doi.org/10.3390/jcdd6030028
Czubryt MP. Cardiac Fibroblast to Myofibroblast Phenotype Conversion-An Unexploited Therapeutic Target. J Cardiovasc Dev Dis. 2019 Aug 16;6(3). doi: 10.3390/jcdd6030028. PMID: 31426390; PMCID: PMC6787657.
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Hypoxia Supports Epicardial Cell Differentiation in Vascular Smooth Muscle Cells through the Activation of the TGFβ Pathway.

Journal of cardiovascular development and disease

Tao J, Barnett JV, Watanabe M, Ramírez-Bergeron D.
PMID: 29652803
J Cardiovasc Dev Dis. 2018 Apr 13;5(2). doi: 10.3390/jcdd5020019.

Epicardium-derived cells (EPDCs) are an important pool of multipotent cardiovascular progenitor cells. Through epithelial-to-mesenchymal-transition (EMT), EPDCs invade the subepicardium and myocardium and further differentiate into several cell types required for coronary vessel formation. We previously showed that epicardial hypoxia...

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Tao J, Barnett JV, Watanabe M, et al. Hypoxia Supports Epicardial Cell Differentiation in Vascular Smooth Muscle Cells through the Activation of the TGFβ Pathway. J Cardiovasc Dev Dis. 2018;5(2)doi: 10.3390/jcdd5020019.
Tao, J., Barnett, J. V., Watanabe, M., & Ramírez-Bergeron, D. (2018). Hypoxia Supports Epicardial Cell Differentiation in Vascular Smooth Muscle Cells through the Activation of the TGFβ Pathway. Journal of cardiovascular development and disease, 5(2), . https://doi.org/10.3390/jcdd5020019
Tao, Jiayi, et al. "Hypoxia Supports Epicardial Cell Differentiation in Vascular Smooth Muscle Cells through the Activation of the TGFβ Pathway." Journal of cardiovascular development and disease vol. 5,2 (2018). doi: https://doi.org/10.3390/jcdd5020019
Tao J, Barnett JV, Watanabe M, Ramírez-Bergeron D. Hypoxia Supports Epicardial Cell Differentiation in Vascular Smooth Muscle Cells through the Activation of the TGFβ Pathway. J Cardiovasc Dev Dis. 2018 Apr 13;5(2). doi: 10.3390/jcdd5020019. PMID: 29652803; PMCID: PMC6023394.
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Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis.

Journal of cardiovascular development and disease

Godby RC, Munjal C, Opoka AM, Smith JM, Yutzey KE, Narmoneva DA, Hinton RB.
PMID: 29552567
J Cardiovasc Dev Dis. 2014 Dec;1(3):237-256. doi: 10.3390/jcdd1030237. Epub 2014 Dec 01.

Aortic valve disease is a burgeoning public health problem associated with significant mortality. Loss of function mutations in NOTCH1 cause bicuspid aortic valve (BAV) and calcific aortic valve disease. Because calcific nodules manifest on the fibrosa side of the...

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Godby RC, Munjal C, Opoka AM, et al. Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis. J Cardiovasc Dev Dis. 2014;1(3):237-256doi: 10.3390/jcdd1030237.
Godby, R. C., Munjal, C., Opoka, A. M., Smith, J. M., Yutzey, K. E., Narmoneva, D. A., & Hinton, R. B. (2014). Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis. Journal of cardiovascular development and disease, 1(3), 237-256. https://doi.org/10.3390/jcdd1030237
Godby, Richard C, et al. "Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis." Journal of cardiovascular development and disease vol. 1,3 (2014): 237-256. doi: https://doi.org/10.3390/jcdd1030237
Godby RC, Munjal C, Opoka AM, Smith JM, Yutzey KE, Narmoneva DA, Hinton RB. Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis. J Cardiovasc Dev Dis. 2014 Dec;1(3):237-256. doi: 10.3390/jcdd1030237. Epub 2014 Dec 01. PMID: 29552567; PMCID: PMC5856658.
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Midkine's Role in Cardiac Pathology.

Journal of cardiovascular development and disease

Woulfe KC, Sucharov CC.
PMID: 28920060
J Cardiovasc Dev Dis. 2017 Sep;4(3). doi: 10.3390/jcdd4030013. Epub 2017 Aug 24.

Midkine (MDK) is a heparin-binding growth factor that is normally expressed in mid-gestational development mediating mesenchymal and epithelial interactions. As organisms age, expression of MDK diminishes; however, in adults, MDK expression is associated with acute and chronic pathologic conditions...

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Woulfe KC, Sucharov CC. Midkine's Role in Cardiac Pathology. J Cardiovasc Dev Dis. 2017;4(3)doi: 10.3390/jcdd4030013.
Woulfe, K. C., & Sucharov, C. C. (2017). Midkine's Role in Cardiac Pathology. Journal of cardiovascular development and disease, 4(3), . https://doi.org/10.3390/jcdd4030013
Woulfe, Kathleen C, and Sucharov, Carmen C. "Midkine's Role in Cardiac Pathology." Journal of cardiovascular development and disease vol. 4,3 (2017). doi: https://doi.org/10.3390/jcdd4030013
Woulfe KC, Sucharov CC. Midkine's Role in Cardiac Pathology. J Cardiovasc Dev Dis. 2017 Sep;4(3). doi: 10.3390/jcdd4030013. Epub 2017 Aug 24. PMID: 28920060; PMCID: PMC5599136.
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Showing 1 to 12 of 520 entries